Cell Death Dis

ISSN	2041-4889
ISSN-L	2041-4889
IUID	605c8d8a88004d4182a0a320cccf1429
Modified	2020-11-27T13:14:08.724Z
Created	2017-10-17T07:53:20.727Z

Year 2024

PubMed DOI Crossref

SETD8 inhibition targets cancer cells with increased rates of ribosome biogenesis.

Murga M, Lopez-Pernas G, Soliva R, ..., Orozco M, Fernández-Capetillo O

Cell Death Dis 15 (9) 694 [2024-09-28; online 2024-09-28]

Chemical Biology Consortium Sweden (CBCS) [Collaborative] Global Proteomics and Proteogenomics [Service]

PubMed DOI Crossref

NOS2-derived low levels of NO drive psoriasis pathogenesis.

Köhler I, Bivik Eding C, Kasic NK, Verma D, Enerbäck C

Cell Death Dis 15 (6) 449 [2024-06-26; online 2024-06-26]

Clinical Genomics [Service] Clinical Genomics Linköping [Service]

PubMed DOI Crossref

LAPTM4B counteracts ferroptosis via suppressing the ubiquitin-proteasome degradation of SLC7A11 in non-small cell lung cancer.

Yan R, Liu D, Guo H, ..., Blom T, Zhou K

Cell Death Dis 15 (6) 436 [2024-06-20; online 2024-06-20]

Swedish Metabolomics Centre (SMC) [Service]

Year 2023

PubMed DOI Crossref

Proteome integral solubility alteration high-throughput proteomics assay identifies Collectin-12 as a non-apoptotic microglial caspase-3 substrate.

Grabert K, Engskog-Vlachos P, Škandík M, ..., Joseph B, Cheray M

Cell Death Dis 14 (3) 192 [2023-03-11; online 2023-03-11]

Chemical Proteomics [Collaborative]

Year 2022

PubMed DOI Crossref

Loss of SNAI1 induces cellular plasticity in invasive triple-negative breast cancer cells.

Tsirigoti C, Ali MM, Maturi V, Heldin CH, Moustakas A

Cell Death Dis 13 (9) 832 [2022-09-28; online 2022-09-28]

Bioinformatics Support for Computational Resources [Service] NGI Short read [Service] NGI Uppsala (SNP&SEQ Technology Platform) [Service] National Genomics Infrastructure [Service]

Year 2021

PubMed DOI Crossref

Inhibition of the ubiquitin-proteasome system by an NQO1-activatable compound.

Giovannucci TA, Salomons FA, Haraldsson M, ..., Johnsen JI, Dantuma NP

Cell Death Dis 12 (10) 914 [2021-10-06; online 2021-10-06]

CRISPR Functional Genomics [Service] Chemical Biology Consortium Sweden (CBCS) [Collaborative] Global Proteomics and Proteogenomics [Service] NGI Stockholm (Genomics Applications) [Service] NGI Stockholm (Genomics Production) [Service] National Genomics Infrastructure [Service]

PubMed DOI Crossref

Super-resolution microscopy reveals that Na+/K+-ATPase signaling protects against glucose-induced apoptosis by deactivating Bad.

Bernhem K, Fontana JM, Svensson D, ..., Brismar H, Aperia A

Cell Death Dis 12 (8) 739 [2021-07-27; online 2021-07-27]

Integrated Microscopy Technologies Stockholm [Technology development]

PubMed DOI Crossref

Genome-wide CRISPR screens reveal synthetic lethal interaction between CREBBP and EP300 in diffuse large B-cell lymphoma.

Nie M, Du L, Ren W, ..., Zhang F, Pan-Hammarström Q

Cell Death Dis 12 (5) 419 [2021-04-28; online 2021-04-28]

Bioinformatics Support for Computational Resources [Service]

Year 2019

PubMed DOI Crossref

BET and Aurora Kinase A inhibitors synergize against MYCN-positive human glioblastoma cells.

Čančer M, Drews LF, Bengtsson J, ..., Weishaupt H, Swartling FJ

Cell Death Dis 10 (12) 881 [2019-11-21; online 2019-11-21]

Bioinformatics Support for Computational Resources [Service] NGI Uppsala (Uppsala Genome Center) [Service] National Genomics Infrastructure [Service]

PubMed DOI Crossref

Correction: APR-246 reactivates mutant p53 by targeting cysteines 124 and 277.

Zhang Q, Bykov VJN, Wiman KG, Zawacka-Pankau J

Cell Death Dis 10 (10) 769 [2019-10-10; online 2019-10-10]

Protein Science Facility (PSF) [Service]

PubMed DOI Crossref

Combining ERBB family and MET inhibitors is an effective therapeutic strategy in cutaneous malignant melanoma independent of BRAF/NRAS mutation status.

Das I, Wilhelm M, Höiom V, ..., Tuominen R, Egyhàzi Brage S

Cell Death Dis 10 (9) 663 [2019-09-10; online 2019-09-10]

NGI Stockholm (Genomics Applications) [Service] NGI Stockholm (Genomics Production) [Service] NGI Uppsala (Uppsala Genome Center) [Service] National Genomics Infrastructure [Service]

Year 2018

PubMed DOI Crossref

Metabolic reprogramming of acute lymphoblastic leukemia cells in response to glucocorticoid treatment.

Dyczynski M, Vesterlund M, Björklund AC, ..., Tamm KP, Nilsson R

Cell Death Dis 9 (9) 846 [2018-08-28; online 2018-08-28]

Global Proteomics and Proteogenomics [Collaborative]

PubMed DOI Crossref

A patient-derived xenograft pre-clinical trial reveals treatment responses and a resistance mechanism to karonudib in metastatic melanoma.

Einarsdottir BO, Karlsson J, Söderberg EMV, ..., Helleday T, Nilsson JA

Cell Death Dis 9 (8) 810 [2018-07-24; online 2018-07-24]

Bioinformatics Support for Computational Resources [Service] NGI Stockholm (Genomics Applications) [Service] NGI Stockholm (Genomics Production) [Service] National Genomics Infrastructure [Service]

PubMed DOI Crossref

BAP1 induces cell death via interaction with 14-3-3 in neuroblastoma.

Sime W, Niu Q, Abassi Y, ..., Fu H, Massoumi R

Cell Death Dis 9 (5) 458 [2018-05-01; online 2018-05-01]

Structural Proteomics [Service]

Year 2017

PubMed DOI Crossref

Silencing FLI or targeting CD13/ANPEP lead to dephosphorylation of EPHA2, a mediator of BRAF inhibitor resistance, and induce growth arrest or apoptosis in melanoma cells.

Azimi A, Tuominen R, Costa Svedman F, ..., Hansson J, Egyhazi Brage S

Cell Death Dis 8 (8) e3029 [2017-08-31; online 2017-08-31]

Clinical Proteomics Mass spectrometry [Service] Global Proteomics and Proteogenomics [Service] NGI Stockholm (Genomics Applications) [Service] NGI Stockholm (Genomics Production) [Service] NGI Uppsala (Uppsala Genome Center) [Service] National Genomics Infrastructure [Service]