Carcinogenesis

PubMed DOI Crossref

Invasive cervical tumors with high and low HPV titer represent molecular subgroups with different disease etiology.

Cui T, Enroth S, Ameur A, ..., Lindquist D, Gyllensten U

pii

Carcinogenesis - (-) - [2018-12-28; online 2018-12-28]

NGI Uppsala (Uppsala Genome Center) [Collaborative] National Genomics Infrastructure [Collaborative]

PubMed DOI Crossref

Analysis of the genetic architecture of susceptibility to cervical cancer indicates that common SNPs explain a large proportion of the heritability.

Chen D, Cui T, Ek WE, ..., Wang H, Gyllensten U

pii

Carcinogenesis 36 (9) 992-998 [2015-09-00; online 2015-06-06]

NGI Uppsala (SNP&SEQ Technology Platform) National Genomics Infrastructure

PubMed DOI Crossref

Systematic investigation of contribution of genetic variation in the HLA-DP region to cervical cancer susceptibility.

Chen D, Gyllensten U

pii

Carcinogenesis 35 (8) 1765-1769 [2014-08-00; online 2014-04-17]

NGI Uppsala (SNP&SEQ Technology Platform) [Service] National Genomics Infrastructure [Service]

PubMed DOI Crossref

WNT5A-mediated β-catenin-independent signalling is a novel regulator of cancer cell metabolism.

Sherwood V, Chaurasiya SK, Ekström EJ, ..., James P, Andersson T

pii pmc

Carcinogenesis 35 (4) 784-794 [2014-04-00; online 2013-12-03]

Tissue Profiling

PubMed DOI Crossref

Estrogen receptor β expression induces changes in the microRNA pool in human colon cancer cells.

Edvardsson K, Nguyen-Vu T, Kalasekar SM, ..., Gustafsson JÅ, Williams C

pii

Carcinogenesis 34 (7) 1431-1441 [2013-07-00; online 2013-02-26]

Tissue Profiling